SARS-CoV-2 spike protein interacts with and activates TLR41

$ 27.50

5 (646) In stock

Biomedicines, Free Full-Text

Frontiers Cryo-electron microscopy in the fight against COVID-19—mechanism of virus entry

SARS-CoV-2 spike protein induces a differential monocyte activation that may contribute to age bias in COVID-19 severity

Innate immunity: the first line of defense against SARS-CoV-2. - Abstract - Europe PMC

Aptamer blocking S-TLR4 interaction selectively inhibits SARS-CoV-2 induced inflammation

SARS-CoV-2 Spike protein is not pro-inflammatory in human primary macrophages: endotoxin contamination and lack of protein glycosylation as possible confounders

SARS-CoV-2 spike protein induces a differential monocyte activation that may contribute to age bias in COVID-19 severity

SARS-CoV-2 spike and nucleocapsid proteins fail to activate human dendritic cells or γδ T cells

SARS-CoV-2 Spike protein induces TLR4-mediated long-term cognitive dysfunction recapitulating post-COVID-19 syndrome in mice - ScienceDirect

Viruses, Free Full-Text

Interferon antagonists encoded by SARS-CoV-2 at a glance

Frontiers Mechanisms of TLR4-Mediated Autophagy and Nitroxidative Stress

SARS-CoV-2 spike protein induces inflammation via TLR2-dependent activation of the NF-κB pathway

Selectively expressing SARS-CoV-2 Spike protein S1 subunit in cardiomyocytes induces cardiac hypertrophy in mice